الفهرس 
IntroductionOnly 14 pages are availabe for public view
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Abstract
The correlation between coronary artery ectasia (CAE) and coronary artery disease (CAD) implies that CAE could potentially be considered as a form of CAD. The precise etiology of coronary artery ectasia (CAE) remains uncertain; nonetheless, it is postulated to entail arterial remodeling as a result of localized plaque proliferation.
In the pathogenesis of atherosclerosis, inflammation assumes a pivotal role, with Hs-CRP being intimately implicated in the atherosclerotic cascade. Other biomarkers like big ET-1,serum uric acid, high-sensitivity C-reactive protein (Hs-CRP) have been found to be correlated with an augmented likelihood of subsequent cardiovascular incidents. Elevated levels of high-sensitivity C-reactive protein (Hs-CRP),big ET-1,Serum UA, LDL, cholesterol, were observed in patients diagnosed with coronary artery ectasia (CAE) when compared to persons with unobstructed coronary arteries, suggesting a potential involvement of inflammatory and atherosclerotic processes in the development of CAE.
Some ratios have been investigated at this study like Big Endothelin 1/HSCRP ratio, HSCRP/serum uric acid ratio, Big Endothelin 1 / serum uric acid ratio, HSCRP/Albumin ratio, LDL/HDL ratio and was found that it were significant clinical differences associated with CAE. Indicating its clinical importance as a predictors for CAE
The investigation of inflammatory biomarkers in CAE exhibits potential in enhancing our comprehension of the ailment and its clinical administration. Clinicians have the opportunity to identify individuals at elevated risk, guide treatment decisions, and potentially develop focused medicines by means of detecting and monitoring certain biomarkers. However, additional study may be required to validate these findings and ascertain the clinical efficacy of these biomarkers in the context of CAE.