الفهرس 
The fourth Universal Definition of Myocardial InfarctionOnly 14 pages are availabe for public view
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Abstract
D-dimer soluble fragments derived from fibrin reflect extent of coagulation, fibrinolysis activation and thrombosis in aortic dissection, peripheral artery disease or venous thromboembolism.(1,2) increased in some acute myocardial infarction (AMI) patients, its potential use as a diagnostic biomarker for AMI remains controversial(3-5) partly because extent of plaque rupture and coronary thrombosis is relatively small compared with that in aortic dissection and venous thromboembolism (6). Although increased plasma D-dimer level among AMI patients is associated with post-interventional coronary no-reflow phenomenon(7-10), which in turn is associated with poor prognosis after ST-segment elevation myocardial infarction (STEMI) 11, studies on the association between D-dimer and prognosis in STEMI patients have yielded inconsistent, results(7-12). During primary percutaneous coronary intervention (PCI), some STEMI patients show angiographically evident thrombus (AET) and no-reflow phenomenon, and the predictive value of increased D-dimer level for adverse outcomes in this subgroup has not been investigated. This study will evaluate the possible association between increased D-dimer level and prognosis in STEMI patients.