الفهرس 
Acknowldgements
Problems associated with the delivery of IDMsOnly 14 pages are availabe for public view
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Abstract
This study aimed to evaluate all full-term infants of diabetic mother admitted in NICU at Assiut University Children Hospital for the presence of hypertrophic cardiomyopathy and follow up of these cases after 6 months for recovery. This descriptive study was conducted on 50 infants of diabetic mother aged 37 – 39 wks. and admitted in NICU. All IDMs admitted in NICU were undergo maternal history, birth history, examination (general appearance, vital Signs, head, and neck), Investigations (serial blood glucose, chest radiographs, echocardiography). Summary of our results:
• HCM found in 34% of studied cases. Only 2% of cases still has a picture of HCM after 6 months. Regarding birth history, it was observed that males contributed 40% of the cases and females contributed 60% of the cases. Gestational age of the studied cases ranged from 37-39 wks. Normal vaginal delivery occurred only in 10% of the cases. In 90% of the cases lower segment cesarean section was performed. Regarding maternal history, gestational diabetes found in 40% of mothers, type 1diabetes found in 16% of mothers, and type 2 diabetes found in 44% of mothers. 38% of mothers controlled on diet, 62% controlled on insulin therapy. In general, 84% of mothers controlled, and 16% uncontrolled. 88% of mothers have only DM without HTN or other diseases and 2% have HTN. 88% of mothers don’t have previous macrosomic infant, and 12% have macrosomic infants. HCM occur in 40.9% in infants their mothers have gestational DM, 25% in infants their mothers have type 1 DM, and 30% in infants their mothers have type 2 DM. according to method of control HCM occur in 42.1% of cases controlled with diet, 29% of cases controlled with insulin . HCM found in 35.7% of infants with controlled mothers , and in 25% of infants with uncontrolled mothers . HCM found in 33.3 of macrosomic infants and 34.1in non macrosomic infants. Comparison between cardiorespiratory according to HCM showed that HCM occur in 66.7% in infants with RD1, in 46.2% in infants with RD2, and 25% in infants with no RD. HCM found in 12.5% in infants with cyanosis and 38.1%in infants without cyanosis. HCM found in 26.3% in infants had hypoglycemia (RBS<45) at 0 hr., and 38.7% in infants with normal blood glucose level (RBS>45) at 0 hr. HCM found in 30.8% of infants with cardiomegaly in chest radiograph, and 35.1of infants with normal chest radiograph. Comparison between ECHO cardio graphic finding of the studies cases according to HCM showed that PFO in 20.8% of cases, ASD in 33.3% of cases, VSD in 14.3% of cases, PDA in 14.3%of cases, TR in 40% of cases, MR in 25% of cases. Hypertrophic cardiomyopathy in newborns can be the clinical expression of different underlying disorders. We aimed to show the importance both to reassess maternal, birth history, clinical examination, cardiorespiratory, Chest radiograph and ECHO cardiographic finding in order to address the correct differential diagnosis. Furthermore it is important to continue a regular cardiologic follow-up for this pathology with neonatal onset to prevent a poor prognosis.